Ventricular tachycardia (abbreviated: VT) is a quick heartbeat (tachycardia) occurring from the ventricles (Lat. ventriculus ventricle) coronary heart. Ventricular tachycardia is among the so-called. periarestne arrhythmia, and is also any existence intimidating situation that could result of ventricular fibrillation, asystole or perhaps sudden loss of life and yes it demands ventricular tachycardia therapy.

Ventricular tachycardia would be the morphology that is certainly identified by electrocardiography, may be divided directly into:

Monomorphic ventricular tachycardia -- almost all heartbeat (QRS complexes) usually are of the identical shape in every one of the ECG leads.
polymorphic ventricular tachycardia -- on the ECG check modifications from the model of the particular QRS defeat for you to defeat.

Ventricular tachycardia signs and symptoms usually are about three or maybe more bests in a very line through the ventricles, that have frequencies in excess of 100 bests and min. In line with the timeframe of VT may be divided directly into:
ventricular tachycardia treatmentVentricular tachycardia (non-sustained) -- would be the name intended for about three or maybe more bests of ventricular rate above 100 bests and instant, which in turn persists under 25 a few moments in addition to stops in an instant
Ventricular tachycardia (sustained) -- is a ventricular tachycardia sustained extended in comparison with 25 a few moments, irrespective of whether a few process is usually interrupted or perhaps quit in an instant.

Monomorphic ventricular tachycardia is usually seen as the particular ECG from the identical varieties of bests, an increase of giving pulses using a core through the still left or perhaps proper ventricle or perhaps because of round currents (reentry) from the ventricles. Usually comes about because of scarring from the myocardium immediately after myocardial infarction.

Polymorphic ventricular tachycardia is frequently because of abnormalities of ventricular repolarization of muscle fibres. Predisposition repolariziaciji abnormalities are generally the particular ECG manifests because QT prolongation, which may be obtained or perhaps congenital. Leads to incorporate congenital long QT predicament, in addition to received mainly linked to the particular poisonous side effects of prescription drugs or perhaps electrolyte disturbances. The actual possible effect regarded antiarrhythmics (sotalol, amiodarone) on QT prolongation, however it is well know many prescription drugs and also the repeated work with including antibiotics, antihistamines may possibly prolong the particular QT phase.

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